NAD+ (Nicotinamide Adenine Dinucleotide) is an essential compound that is integral to cellular health. It plays a pivotal role in cellular metabolism, helping to ensure well-balanced cellular function and energy production. Research indicates that NAD+ levels decline with age, which significantly impacts the aging process by reducing cellular vitality and function.

Selerb's innovative NAD+ formula is designed to counteract these effects by boosting the body's NAD+ levels along with other vital ingredients. Utilizing advanced technology, this formula enhances the absorption of Nicotinamide Riboside (NR) — a precursor to NAD+ — into the cellular structure. Through rigorous research and testing, Selerb has also identified complementary components that exhibit synergistic effects with NR, enhancing the anti-aging benefits of the formula.

The enhanced NAD+ technology from Selerb not only replenishes the NAD+ necessary for optimal cellular energy but also improves metabolic functions and mitigates the symptoms of cellular aging. Regular use of this product can lead to revitalized cellular health, increased vitality, restored skin luminosity, and a marked improvement in overall physical wellness.

Studies have shown that maintaining elevated NAD+ levels can help delay some aspects of aging and improve metabolic health. For instance, research by Verdin (2015) emphasizes the role of NAD+ in regulating protein interactions and energy metabolism, which are critical in aging and chronic disease. Further studies by Rajman, Chwalek, & Sinclair (2018) explore the potential of NAD+ supplementation in promoting health and extending lifespan by enhancing mitochondrial function.

Moreover, a study by Yoshino et al. (2018) demonstrated that NAD+ precursors could improve glucose metabolism and reduce signs of aging in mice, suggesting potential applications in humans. Camacho-Pereira et al. (2016) also contributed to the understanding of how NAD+ impacts cellular metabolism and stress responses, reinforcing the importance of maintaining NAD+ levels through dietary or supplementary means.

References:

• Verdin, E. (2015). NAD+ in aging, metabolism, and neurodegeneration. Science, 350(6265), 1208-1213.
• Rajman, L., Chwalek, K., & Sinclair, D.A. (2018). Therapeutic potential of NAD-boosting molecules: the in vivo evidence. Cell Metabolism, 27(3), 529-547.
• Yoshino, J., Mills, K.F., Yoon, M.J., & Imai, S. (2018). Nicotinamide mononucleotide, a key NAD+ intermediate, treats the pathophysiology of diet- and age-induced diabetes in mice. Cell Metabolism, 14(4), 528-536.
• Camacho-Pereira, J., Tarragó, M.G., Chini, C.C.S., Nin, V., Escande, C., Warner, G.M., et al. (2016). CD38 dictates age-related NAD decline and mitochondrial dysfunction through an SIRT3-dependent mechanism. Cell Metabolism, 23(6), 1127-1139.